Ca2+ leak—What is it? Why should we care? Can it be managed?Boyden, Penelope A.Smith, Godfrey L.HEART RHYTHM
Ca2+ leak-What is it? Why should we care? Can it be managed?For arrhythmia triggers that are secondary to dysfBoydenUniv Boyden,Penelope,A.,... - 《Heart Rhythm the Official Journal of the Heart Rhythm Society》 被引量: 0发表: 2018年 Ca2+ leak-What is it? Why should we care? Can...
sarcoplasmic reticulum (SR), and mitochondrial Ca2+mishandling in diabetic cardiomyopathy and how these alterations may be involved in the decrease in contractility, arrhythmias, and apoptosis observed that may finally end in overt HF. In the last part of the review, we will depict what is known ...
6. Although it is not possible to assign these peaks to particular residues of the protein, it is possible to say what type of residues they represent. The peak in the aromatic region (Fig. 6A) is likely to be from a histidine proton, since it shows a characteristic shift on change of...
(vehicle n = 3, 136 NMJs) and in TRAM-34-treated hSOD1G93A mice this value drops to 0.96 ± 0.06 (TRAM-34n = 4, 135 NMJs), not dissimilar to what was observed in non-tg age matched mice (0.4 ± 0.06 per NMJ, non-tg n = 3, 148 NMJs) (see ...
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Memory built in conjunction with a stressor is privileged: Reconsolidation-resistant memories in the crab Neohelice. Brain Res. Bull. 157, 108–118 (2020). Article CAS PubMed Google Scholar Pedreira, M. E., Perez-Cuesta, L. M. & Maldonado, H. Mismatch between what is expected and what...
While actin is clearly present around synaptic vesicle clusters, it is less clear what role it plays in synaptic transmission. In this study we have used phalloidin, both as a marker of presynaptic terminals, and also as an inhibitor of actin depolymerization selectively applied to the presynaptic...
So if Ca2+ goes down by this much, I'd subtract it from 0.0062M but this is a negative value?!? :S Logged XGen Full Member Posts: 127 Mole Snacks: +9/-4 Re: How to calculate what percentage of Ca2+ remains unprecipitated? « Reply #5 on: March 04, 2012, 10:05:55 PM ...
In this way RyRs-dependent Ca2+ release functionally couples plasma membrane Cav1 channels to mitochondria which, similarly to what has been described in cardiac cells [124], are docked to the ER at MAMs. If continuous Ca2+ entry sustains dopamine secretion and mitochondrial metabolism, at the ...